Cholesterol 7α-hydroxylase Overview
Cholesterol 7α-hydroxylase (CYP7A1) is a liver-specific microsomal cytochrome P450 enzyme that catalyzes the initial and rate-limiting step in the classic pathway of bile acid biosynthesis, converting cholesterol to 7α-hydroxycholesterol[7][1]. It plays a central role in maintaining cholesterol and bile acid homeostasis, affecting lipid metabolism and nutrient absorption. CYP7A1 is tightly regulated at the transcriptional and post-transcriptional levels by nuclear receptors (FXR, LXR, HNF4α, SHP, FOXO1, LRH-1) and by microRNAs such as miR-33a and miR-122a. Therapeutic modulation of CYP7A1 is explored in managing hyperlipidemia, NAFLD, obesity, and related metabolic disorders[1][2][3][6][7].
Mechanism of Action
Transcriptional repression or activation: FXR agonists suppress CYP7A1 transcription via nuclear receptor signaling. miRNA-mediated post-transcriptional regulation: miR-33a, miR-122a, and miR-422a modulate CYP7A1 mRNA stability and translation in hepatocytes. Ligand activation: Nuclear receptors (LXR, FXR, HNF4α, SHP, FOXO1, LRH-1) bind the CYP7A1 promoter and regulate activity in response to metabolites and hormones.
Biological Functions
Disease Associations
Safety Considerations
- Risk of cholestasis or hepatotoxicity if bile acid synthesis becomes dysregulated
- Potential for altered lipid absorption and fat-soluble vitamin deficiency
- Possible interaction with other metabolic pathways via nuclear receptor cross-talk
Interacting Drugs
Associated Biomarkers
Biomarker |
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Serum cholesterol and triglyceride levels |
Bile acid pool size and composition |
Hepatic CYP7A1 mRNA or protein expression |