RNA, 7SL, cytoplasmic 309, pseudogene (RN7SL309P)

**RN7SL309P** is a human pseudogene—a nonfunctional DNA segment derived from the reverse integration of 7SL RNA into the genome[5][6]. It represents a non-functional, DNA-encoded version of the 7SL RNA molecule, which is a key component of the signal recognition particle (SRP), vital for directing newly synthesized proteins to the endoplasmic reticulum and controlling protein translation and targeting[7][8]. Pseudogenes like RN7SL309P do **not encode active proteins or functional RNA** and are generally considered non-coding and nonfunctional[1][6]. RN7SL pseudogenes, like RN7SL309P, are truncated, mutated, or otherwise inactivated copies of the parental 7SL RNA gene[5][6]. They are generally not transcribed or translated into any functional products, nor do they serve as viable therapeutic targets. While some pseudogenes have regulatory potential (e.g., acting as miRNA sponges or producing small interfering RNAs), there is no current evidence or consensus supporting a functional or disease-associated role for RN7SL309P[1][4][5]. Pseudogenes typically lack direct biological functions; however, some may act as competing endogenous RNAs (ceRNAs) or produce small RNAs that can potentially regulate parental gene expression—this is primarily established for other well-studied pseudogenes, with sparse evidence for RN7SL pseudogenes directly[4]. No disease association, as RN7SL309P is not expressed as a functional gene or implicated in pathology under current knowledge[1][5]. No known drugs interact with RN7SL309P, as it is not a protein or viable RNA target for pharmacology[1]. In summary, **RN7SL309P is not a drug target, has no known biological or disease function, and introduction of this entry as a therapeutic target or biomarker would be scientifically incorrect and misleading**[1][5][6].

Other names

RN7SL309PRNA, 7SL, cytoplasmic 309, pseudogene