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SH3GL1P3 is a human pseudogene, listed as “SH3 domain containing GRB2 like 1, endophilin A2 pseudogene 3” and also referred to as CNSA-P3[4]. Pseudogenes are nonfunctional segments of DNA that are similar to known genes but are typically not transcribed into functional proteins. There is no evidence in scientific literature or curated bioinformatics sources that SH3GL1P3 produces a functional receptor, enzyme, transporter, or any other typical drug target. It is not considered a therapeutic target, and there is no known interaction with drugs, nor does it have identifiable roles in physiology, disease, or as a biomarker. The “SH3GL1P3” and “CNSA-P3” refer to the same sequence and do not denote a protein or druggable receptor. No function, disease association, or pharmacological profile exists for this entry. The abbreviation “CNSA-P3” appears to be a synthetic or database-specific alias for this pseudogene entry with no link to therapeutic targeting, signaling, or canonical protein biology. There is no relationship with p3 peptide, amyloid pathology, Alzheimer's disease, or any active drug discovery target[1][2][3]. If your intent was to interrogate the p3 peptide (amyloid β (Aβ) 17–40/42 fragment), this is a distinct molecule entirely and not related to SH3GL1P3. SH3GL1P3 (CNSA-P3) is a pseudogene, not a therapeutic target, and has no documented function in biology or medicine[4]. There are no known molecular interactions, disease roles, or pharmacological relevance. This entry is incorrectly presented as a “target”; it is simply a noncoding pseudogene.
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