BCL2 interacting protein 3 pseudogene 37 (BNIP3P37)

BCL2 interacting protein 3 pseudogene 37 (BNIP3P37) is annotated as a *processed pseudogene* in the human genome. Unlike its parental gene BNIP3, which plays a well-characterized role in apoptosis and autophagy, BNIP3P37 **does not encode a functional protein** and is not currently associated with any known molecular function, biological process, or clinical role[1][5]. There are no data supporting its use as a therapeutic target, biomarker, or pharmacological receptor or enzyme. Pseudogenes like BNIP3P37 are typically “defunct” genes that have lost their ability to encode proteins, although some pseudogenes can be transcribed and, in rare cases, regulate gene expression via noncoding RNA mechanisms[5]. As of current knowledge, no such regulatory or disease-related function is reported for BNIP3P37. **Notes on incorrectness**: - BNIP3P37 is not a targetable molecule, does not code for a druggable protein, and should not be confused with its parental gene *BNIP3*, which is a member of the BCL2 protein family involved in cell death pathways[3][6]. - If you are looking for the functional protein, refer to **BCL2/adenovirus E1B 19kDa interacting protein 3 (BNIP3)**, not BNIP3P37[3]. **Summary**: BNIP3P37 is a pseudogene, **not a therapeutic target**, and has no currently known biological or clinical significance as a molecule or receptor[1][5].

Other names

BCL2/adenovirus E1B 19kDa interacting protein 3 pseudogene 37BNIP3P37