Blood glucose level regulation

"Blood glucose level regulation" is **not** the name of a specific molecule, receptor, enzyme, or drug target. Instead, it refers to the complex physiological process by which the body maintains blood sugar within a narrow range. This process involves multiple hormones—primarily **insulin** and **glucagon**, both secreted by the pancreas—as well as various organs such as the liver, muscle tissue, adipose tissue, and kidneys[1][2][3][5]. Insulin lowers blood glucose by promoting cellular uptake and storage as glycogen or fat; glucagon raises it by stimulating glycogen breakdown in the liver[1][3]. Additional regulatory factors include somatostatin from pancreatic delta cells[1]. The molecular effectors involved in this system include hormone receptors (e.g., insulin receptor), enzymes involved in glycogen metabolism, and several classes of glucose transporters such as GLUT1–4 and SGLT proteins[2]. Drugs that interact with these pathways include exogenous insulin preparations for diabetes management or glucagon injections for severe hypoglycemia[3]. Because "blood glucose level regulation" describes an entire physiological pathway rather than an individual molecular entity or therapeutic target, it should not be considered a canonical drug target. For structured data purposes—such as those used in pharmacology databases—the correct approach would be to specify one of its key components like "Insulin receptor," "Glucagon receptor," "Sodium-glucose cotransporter 2," etc., depending on context. In summary: **Blood glucose level regulation is not itself a molecule or therapeutic target but rather describes an essential metabolic control system involving many targets.** If you need information about specific molecules within this pathway—such as insulin receptor or glucagon-like peptide 1 receptor—please specify which component you are interested in.[1][2][3]

Other names

Blood glucose regulationBlood sugar regulationGlucose homeostasis

The process involves insulin lowering blood glucose by promoting cellular uptake and storage as glycogen or fat, and glucagon raising it by stimulating glycogen breakdown in the liver.