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The term "Cancer cell mitosis process" refers broadly to the sequence of events by which cancer cells undergo division and proliferation through mitosis. This is not a single molecular target but an overarching biological phenomenon involving numerous regulatory proteins and pathways. In healthy tissue, mitosis is tightly regulated by checkpoints and signaling pathways that ensure proper DNA replication and chromosome segregation. In cancer, these controls are frequently disrupted due to mutations in oncogenes (such as Ras and Myc) and tumor suppressors (such as p53), leading to uncontrolled proliferation[2][3]. The dysregulation of genes controlling various phases of the cell cycle—particularly those governing entry into S phase and progression through G2/M checkpoint—is central to tumor growth[1]. While many anti-cancer therapies aim at inhibiting key molecules within this pathway (e.g., cyclin-dependent kinases), "cancer cell mitosis process" itself is too broad for use as a canonical drug target or biomarker; instead, research focuses on its constituent molecular components. Note: This entry does *not* correspond to a discrete molecule/receptor/protein but rather describes an entire cellular program. For structured data purposes, it should be flagged as incorrect for use as a canonical therapeutic target name.
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