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Cellular oxidative pathways refer to the ensemble of biological processes in which cells utilize oxidation-reduction reactions for energy production, metabolism, and signal transduction. The most prominent example is **oxidative phosphorylation**, where nutrients are oxidized in mitochondria, producing ATP and generating ROS as by-products[1][5]. Other pathways include the enzymatic generation and scavenging of ROS in various cell compartments. While these pathways are central to homeostasis, dysregulation leads to **oxidative stress**, contributing to aging, neurodegeneration, cancer, cardiovascular disease, and other disorders, and are thus a focus of many therapeutic approaches[4][6][7].\n\n**Note:** \"Cellular oxidative pathways\" is a functional/system-level concept, not a canonical single drug target, receptor, enzyme, or protein. If you need information about a **specific protein target within these pathways** (e.g., NADPH oxidase, mitochondrial complex I, or a particular ROS enzyme), please clarify.
Scavenging ROS (antioxidants)\n- Inhibiting mitochondrial electron transport (complex inhibitors)\n- Modulating redox-sensitive signaling pathways (e.g., Nrf2 activators)
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