Target intelligence / Profile preview

Eukaryotic translation elongation factor 1 alpha 1 pseudogene 19 (EEF1A1P19)

Target
EEF1A1P19
Molecular classification
Other (Pseudogene)
01

Overview

Eukaryotic translation elongation factor 1 alpha 1 pseudogene 19 (EEF1A1P19) is a human processed pseudogene derived from the EEF1A1 gene family, which encodes one of the main components of the protein synthesis elongation cycle[1][3]. EEF1A1P19 does not encode a functional protein and is not currently implicated in any well-established biological functions or diseases. The broader EEF1A1 gene has many pseudogenes (over 40 in the human genome), and while some EEF1A1 pseudogenes have been investigated for potential roles in gene regulation or cancer, EEF1A1P19 has not been characterized beyond its annotation as a pseudogene[1][3]. As such, it is not considered a therapeutic target, has no known drug interactions, and holds no established value as a biomarker or disease-associated risk factor. Key contextual points: - Pseudogenes like EEF1A1P19 are non-coding DNA sequences resembling known genes but are generally not transcribed or translated into functional proteins[1]. - While the parental gene (EEF1A1) is a critical enzyme for translation and is involved in numerous cellular pathways and diseases, the pseudogene EEF1A1P19 itself has no known function, interaction, or therapeutic relevance[1][4]. - Naming of these pseudogenes is systematic, and individual pseudogenes are rarely assigned alternative aliases unless specifically studied in a biological context[3]. Summary: EEF1A1P19 is a non-coding human pseudogene, one of more than 40 similar elements derived from EEF1A1, and has no known biological function, disease association, or therapeutic importance[1][3][4].

Other names
EEF1A1P19
02

Biological functions

Other (No protein product; potential regulatory function as a pseudogene, but not established)
03

Disease associations

Other (No established disease association for EEF1A1P19 specifically; some EEF1A1 pseudogenes are being explored for their regulatory or disease-linked roles, but EEF1A1P19 is not individually reported in disease context[1])

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