Homo sapiens microRNA 6796-5p (hsa-miR-6796-5p)

Homo sapiens microRNA 6796-5p (hsa-miR-6796-5p) is a small, non-coding RNA molecule that functions as a microRNA, regulating gene expression at the post-transcriptional level by binding to complementary sequences in the 3' untranslated regions (UTRs) of target messenger RNAs (mRNAs), leading to mRNA degradation or inhibition of translation[1][6]. hsa-miR-6796-5p has high-confidence predicted or experimentally validated gene targets involved in immune response, cellular development, movement, proliferation, cell cycle, and cell survival[1][3][5]. It plays a role in the regulation of signaling pathways relevant to diseases such as ischemic stroke (where it regulates matrix metalloproteinase 9 via circSKA3 as a competing endogenous RNA[4]), and potentially in coronavirus infection and pathogenesis by targeting genes like HDAC3, Rab7a, and androgen receptor[1]. The expression of hsa-miR-6796-5p is altered in pathophysiological states, for example in ischemic stroke and possibly in oral cancer[3][4][7]. hsa-miR-6796-5p itself is not a typical "therapeutic target" in the receptor/enzyme/ion channel sense, but may serve as a biomarker or be therapeutically targeted indirectly through the modulation of its expression or interactions[4]. No drugs are listed as interacting directly with hsa-miR-6796-5p.

Other names

miR-6796-5phsa-mir-6796-5phsa-mir-6796