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The \"Host immune response to viral antigens\" is not a single canonical drug target, receptor, or molecule, but rather a complex series of cellular and molecular mechanisms constituting the host’s immune defense against viruses. This includes the recognition of viral antigens by both innate and adaptive immune components (such as dendritic cells, B cells, T cells, and natural killer cells), the production of antiviral antibodies, the activation of cytotoxic T lymphocytes to eliminate infected cells, and the release of cytokines that orchestrate immune responses[1][2][3][5][6][7]. Viral antigens are detected by pattern recognition receptors and presented to lymphocytes, leading to immune memory and, in some cases, the basis for vaccination. Viruses utilize a variety of immune evasion strategies, such as antigenic drift/shift, molecular mimicry, and suppression of host cytokine responses, which complicate the immune response and can contribute to diseases like autoimmunity and persistent infection[1][3][4][5].
Induction of neutralizing antibodies; Cytotoxic T lymphocyte activation to lyse virus-infected cells; Cytokine-mediated antiviral state induction; Enhancement of antigen presentation
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