Immune evasion

Immune evasion refers to the collection of strategies utilized by pathogens (such as viruses, bacteria, and cancer cells) to evade detection, recognition, or elimination by the host immune system. These mechanisms include altering surface proteins, expressing immune-suppressive molecules, downregulating antigen presentation, producing decoy receptors, and mimicking host cells. The ability to evade immunity is a key contributor to persistent infection, cancer progression, and poor response to immunotherapies[2][3][4][5][1]. Reason for "is_incorrect: true": "Immune evasion" does not represent a discrete, actionable therapeutic target such as a receptor, enzyme, or transporter. It is a conceptual process or phenomenon involving multiple molecular players and pathways, not a molecule or protein that can be directly targeted by drugs[2][5][7]. For structured data on therapeutic targets, you would need to specify individual molecular components (e.g., "Programmed cell death protein 1 (PD-1) receptor," "Cytotoxic T-lymphocyte-associated protein 4 (CTLA-4) receptor," "Transforming growth factor beta (TGF-β)") that participate in immune evasion pathways[3][1][6].