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Ocular surface homeostasis support" is not a specific molecule, receptor, or canonical therapeutic target. Instead, it refers to the overall physiological process by which the ocular surface—including the cornea, conjunctiva, and tear film—maintains a stable and healthy environment. This dynamic equilibrium involves an intricate balance among lipid, aqueous, and mucin layers in the tear film to ensure lubrication, protection from environmental stressors (such as dryness or foreign bodies), and optimal vision. Disruption of this homeostatic state can lead to conditions like dry eye disease (DED), characterized by inflammation and instability of the tear film. The concept is central in understanding both normal function and pathology but does not correspond to a single molecular entity that could be directly targeted by drugs[1][2][4].
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