Ribosomal protein L36a pseudogene 17 (RPL36AP17)

Ribosomal protein L36a pseudogene 17 (RPL36AP17) is a non-coding, processed pseudogene related to the *RPL36A* gene, which encodes a structural protein of the 60S ribosomal subunit. Pseudogenes like RPL36AP17 do not produce functional proteins and typically do not have established roles in protein synthesis, cell signaling, disease, or drug interaction. They are remnants of gene duplication or retrotransposition events and are widely considered non-functional in terms of encoding proteins[6][7][9]. RPL36AP17 is not a protein-coding gene, but a processed pseudogene related to the functional RPL36A gene[6][7][9]. The functional parent gene, RPL36A (ribosomal protein L36a), encodes a protein component of the 60S ribosomal subunit, important for protein synthesis[1][3][4]. However, pseudogenes like RPL36AP17 do not encode functional proteins. RPL36AP17 is not a validated receptor, enzyme, transporter, or traditional therapeutic target. There are no known drugs, mechanisms of action, or disease associations directly linked to this pseudogene[7][6]. The presence of "pseudogene" in the name explicitly indicates non-coding status and lack of direct biological or therapeutic function[6][7]. RPL36AP17 is not a druggable or disease-relevant molecular target; rather, it is a genomic pseudogene. If a functional drug or therapeutic mechanism is sought, this entry is incorrect as it refers to a non-protein-coding genomic relic. The confusion may arise from similarity with the true protein-coding gene RPL36A[1][4][6]. "is_incorrect": true because—while real as a genomic element—this "target" does not accord with conventional definitions used in drug discovery or biology for actionable therapeutic molecular entities. If you require information on the functional ribosomal protein, refer instead to RPL36A (ribosomal protein L36a), which is a bona fide protein-coding gene and structural component of ribosomes[1][3][4].

Other names

RPL36A_4_361RPL36AP17ribosomal protein L36a pseudogene 17