Ribosomal protein S29 pseudogene 26 (RPS29P26)

RPS29P26 is a processed pseudogene; it was derived from its parent gene, ribosomal protein S29 (*RPS29*), which encodes a structural protein of the small 40S ribosomal subunit[3][5]. Pseudogenes like RPS29P26 generally do **not encode functional proteins** and are typically **not considered therapeutic targets**, as they do not participate in biological functions such as protein synthesis, signaling, or disease mechanisms[1][6]. Their primary scientific value is for comparative genomics, evolutionary studies, or sequence probe design. There are **multiple processed pseudogenes** of ribosomal protein genes scattered throughout the human genome, including RPS29P26[6]. These pseudogenes arise by retrotransposition and typically do not have biological activity, functional protein expression, or disease causality. As with most ribosomal protein pseudogenes, RPS29P26 is **not a receptor, enzyme, transporter, transcription factor, nor any classical therapeutic target**[1][3][5][6]. There are **no known drugs, biomarkers, disease associations, or therapeutic safety concerns** related to RPS29P26 specifically. **Summary of conventions:** - The gene is **not a functional protein** or recognized target. - It is **incorrect to use RPS29P26 as a therapeutic target** or in drug development contexts. - Processed pseudogene entries like RPS29P26 should be excluded from target lists focused on functional molecules. **Note:** If the intended query was for the parent protein, **Ribosomal protein S29 (RPS29)**, refer to it as a **structural component of the small ribosomal subunit involved in protein synthesis; mutations in RPS29 can cause Diamond-Blackfan anemia**[3][5]. However, **RPS29P26 is a non-functional gene copy (pseudogene) and not a protein-coding or disease-associated gene**[1][6].

Other names

RPS29_20_1655RPS29P26