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Septin 7 pseudogene 11 (SEPTIN7P11) is a genetic locus classified as a pseudogene, most likely arising from gene duplication or retrotransposition of the SEPTIN7 gene[2][4][5]. Pseudogenes like SEPTIN7P11 contain sequence similarity to their functional parental genes but typically include disabling mutations—such as frameshifts, premature stop codons, or the absence of regulatory elements—that prevent them from coding for functional proteins[3][2][5][7]. Though many pseudogenes are regarded as "junk DNA," recent research shows that some have regulatory functions at the RNA level, including modulation of gene expression, serving as microRNA decoys, or contributing to genetic diversity[1][4]. However, there is no evidence in the scientific literature that SEPTIN7P11 has any such regulatory function, protein expression, or relevance to disease or pharmacology. Thus, SEPTIN7P11 is not considered a receptor or therapeutic target, and its annotation as a target is incorrect. If you need structured data on validated therapeutic targets, SEPTIN7P11 should be excluded, and focus placed on functional genes, receptors, enzymes, or other druggable entities.
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