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The skin healing process, commonly termed wound healing or cutaneous repair, is an orchestrated series of biological events that restore skin integrity following injury. It proceeds through classical phases: hemostasis (clot formation), inflammation, proliferation (including cell migration and proliferation, new tissue and matrix deposition, angiogenesis), and tissue remodeling, culminating in resolution or scar formation. Numerous molecular pathways, cell populations (keratinocytes, fibroblasts, immune cells, endothelial cells), and signaling molecules (growth factors, cytokines, proteases) cooperate to ensure effective healing. Chronic wounds, excessive fibrosis, or impaired repair arise from dysregulation of constituent processes. To represent a drug target, you must specify a specific protein, receptor, enzyme, or molecular pathway within the "skin healing process" (for example, "Epidermal growth factor receptor," "Transforming growth factor beta 1," "Matrix metalloproteinase 9," etc.).

Other names

Wound healing processCutaneous repairSkin regeneration

Varies by molecular target; common mechanisms are: Stimulation of cell proliferation and migration; Modulation of inflammation; ECM remodelling; Angiogenesis promotion; Antiscarring/fibrosis inhibition; Immune modulation